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It has been reported that blocking cyclin D1 can induce the cell cycle arrest of human chondrocytes, thereby inducing apoptosis and protecting articular cartilage. Feng et al. reported that blocking cyclin D1 can protect the articular cartilage by inducing chondrocyte cell cycle arrest and initiating apoptosis. To further analyze this mechanism, Feng et al. discovered that cyclin D1 inhibits the apoptosis of chondrocytes by regulating the expression of RARRES1 (retinoic acid receptor responder 1) and CDK8. Feng and colleagues further demonstrated that Smad1 is a prerequisite of RARRES1, and CDK8 is a prerequisite of Smad1. RARRES1 inhibits CDK8, thereby reducing the CDK8-mediated phosphorylation of Smad1. Feng et al. further demonstrated that activation of CDK8 is indispensable for Smad1 phosphorylation (activation). Feng and colleagues concluded that blocking cyclin D1 results in the inhibition of Smad1 phosphorylation, thereby inhibiting RARRES1-mediated CDK8 degradation, and that both RARRES1 and CDK8 are necessary for maintaining the viability of chondrocytes.
In addition, it has been reported that the C-peptide of human insulin can promote the proliferative capacity of art